Color atlas of pathology: pathologic principles, associated by Ursus-Nikolaus Riede, Martin Werner

By Ursus-Nikolaus Riede, Martin Werner

The pocket atlas is a superb reference paintings that provides pathologic findings in all clinically suitable fields, utilizing fine quality photos and concise, good based explanatory texts. pictures contain colour images and micrographs. Electron microscopic photographs are integrated the place helpful. transparent schematic drawings illustrate strategies akin to the process infections, and so forth. textual content packing containers on scientific and morphological points, on problems, and with easy-to-remember mnemonic summaries are trigger in colour.

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Additional info for Color atlas of pathology: pathologic principles, associated diseases, sequela

Sample text

Consequently, there is no zone of vesicular cartilage and no cartilage breakdown zone ( E1 and F). This results in the formation of a broad, shortened area of primary cancellous bone with delayed remodeling into secondary cancellous bone ( E2). The long cortical bones remain short and broad, with caps of epiphyseal cartilage ( D), resulting in dwarfism. However, ligamentous ossification proceeds normally; growth of the sides and top of the skull is normal, although the base of the skull remains small.

Example: Alcoholic hepatitis (p. 144). Note: Mallory bodies are an equivocal sign of toxic liver damage from alcohol abuse. The diseases discussed up to this point are attributable to enzyme deficiencies and functional derangements of certain cellular organelles. The following section examines diseases caused by aberrant synthesis or breakdown processes of the extracellular matrix. They represent dysfunctions of structural metabolism. Example: Parkinson’s disease (p. 114). Riede / Werner, Color Atlas of Pathology © 2004 Thieme All rights reserved.

Consequently, there is no zone of vesicular cartilage and no cartilage breakdown zone ( E1 and F). This results in the formation of a broad, shortened area of primary cancellous bone with delayed remodeling into secondary cancellous bone ( E2). The long cortical bones remain short and broad, with caps of epiphyseal cartilage ( D), resulting in dwarfism. However, ligamentous ossification proceeds normally; growth of the sides and top of the skull is normal, although the base of the skull remains small.

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